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A novel Hox gene promoter fuels the evolution of adaptive phenotypic plasticity in wing eyespots of satyrid butterflies

Abstract

Adaptive phenotypic plasticity allows organisms to display distinct phenotypes in response to variable environments, but little is known about the genomic changes that promote the evolution of plasticity on a macroevolutionary scale. Here, combining tissue-specific transcriptomics, comparative genomics and genome editing, we show that temperature-mediated plasticity in the size of butterfly eyespot wing patterns, a derived seasonal adaptation estimated to have evolved ~60 million years ago at the base of the satyrid clade (~2,700 extant species), is fuelled by the recruitment of a Hox gene Antennapedia (Antp) to eyespot development. In satyrid butterflies, Antp regulates eyespot size in a temperature-dependent manner, increasing plasticity levels. The cooption of Antp to eyespots was driven by the evolution of a novel eyespot-specific promoter in satyrid genomes, which when disrupted in a model satyrid, Bicyclus anynana, reduced plasticity levels. We show that a taxon-specific cis-regulatory innovation in a conserved developmental gene fuelled the evolution of adaptive phenotypic plasticity across a large clade of animals.

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Fig. 1: Tissue-specific transcriptomes generated from a developmental window of plasticity.
Fig. 2: Genes showing trait-specific responses to cues are potential plasticity regulators.
Fig. 3: Antp regulates temperature-mediated eyespot size plasticity in satyrid butterflies.
Fig. 4: A novel promoter activates Antp expression in satyrid eyespots.
Fig. 5: Antp P1 regulates satyrid eyespot size plasticity.
Fig. 6: An evolutionary–developmental model of a classic system of adaptive phenotypic plasticity.

Data availability

All data required for reproducing and extending the study are available in the main text or the Supplementary Information. Raw RNA-seq data for Bicyclus anynana are available under NCBI BioProject PRJNA1268022. Raw genome sequencing and assembly data for Junonia almana are available under NCBI BioProject PRJNA1300591. Lists of differentially expressed genes and enriched Gene Ontology terms generated in the transcriptomic analysis and lists of HCR probe sequences are available via figshare at https://doi.org/10.6084/m9.figshare.c.8026060.v1 (ref. 55). Source data are provided with this paper.

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Acknowledgements

We thank J. Lee for his assistance with the promoter usage analysis. We thank K. Long Tan for helping rear the promoter knockout lines. We acknowledge National Research Foundation (NRF) Singapore award NRF-CRP20-2017-0001 (A.M.), NRF Singapore award NRF-CRP25-2020-0001 (A.M.), NRF Singapore award NRF-NRFI05-2019-0006 (A.M.) and Ministry of Education Singapore award MOE-T2EP30223-0007 (A.M.).

Author information

Authors and Affiliations

Authors

Contributions

Conceptualization: S.T. and A.M. Methodology: S.T., T.D.B. and A.M. Investigation: S.T., B.L., T.D.B. and S.N.M. Visualization: S.T., B.L. and T.D.B. Funding acquisition: A.M. Project administration: S.T. and A.M. Supervision: A.M. Writing—original draft: S.T. and A.M. Writing–review and editing: S.T., T.D.B. and A.M.

Corresponding authors

Correspondence to Shen Tian or Antónia Monteiro.

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The authors declare no competing interests.

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Nature Ecology & Evolution thanks Jennifer Brisson, Sofia Casasa and the other, anonymous, reviewer(s) for their contribution to the peer review of this work. Peer reviewer reports are available.

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Extended data

Extended Data Fig. 1 Temperature-shift experiments identified a prolonged temperature sensitivity window.

Shift schemes in refined temperature-shift experiments are illustrated (left panel). See Methods and Supplementary Table 1 for detailed methods and statistics of the temperature-shift experiments. Ventral Cu1 eyespot areas (middle panel), and temperature sensitivities (right panel, only across the six non-overlapping windows) were quantified and compared across the schemes in a one-way ANCOVA (Methods). n = 35 (WS), 20 (L5-1), 20 (L5-2), 12 (L5-3), 24 (Wr), 23 (PP), 22 (P15), 23 (Wr+PP), 23 (Wr+PP + P15), 24 (L5), 27 (DS). Black dots with error bars represent mean values ± 95CI. Eyespot areas from shift schemes with the same letter are not significantly different from each other, as determined by Tukey’s test. WS, wet season; DS, dry season; L5, 5th instar larva; Wr, wanderer, PP, prepupa; P, pupa.

Source data

Extended Data Fig. 2 Co-staining of a positive eyespot marker sal in J. almana.

Sal was used as a positive eyespot marker in both immunostaining and HCR. For immunostaining and HCR, n = 3 replicates. Scale bar: 100 microns.

Extended Data Fig. 3 Calculation of Antp protein eyespot expression diameter.

Size of the Antp protein expression domain in eyespots was calculated as eyespot expression diameter. For each ventral hindwing eyespot (here we use Cu1 eyespot as an example), eyespot expression diameter was calculated as the mean of the two diameters of the Antp protein expression domain (a and b), one (a) was parallel to the proximal-distal axis of the sector (outlined in dotted lines), while the other (b) perpendicular to it. The distance between M3 and Cu1 eyespot centers (c) was constantly used as a hindwing size proxy, and entered as a covariate in statistical analyses. Scale bar: 100 microns.

Extended Data Fig. 4 Representative phenotypes of Antp mKO crispants in B. anynana.

A dotted line separates left and right sides of the same individual. Purple arrowheads denote mutant phenotypes.

Extended Data Fig. 5 Representative phenotypes of Antp mKO crispants in M. mineus.

A dotted line separates left and right sides of the same individual. Purple arrowheads denote mutant phenotypes.

Extended Data Fig. 6 Conservation of sequences and transcriptional activities of Antp promoters across B. anynana and two non-satyrid butterflies.

Antp transcript annotation and sequence conservation around the six Antp promoter regions in B. anynana and two non-satyrids, V. cardui and J. coenia. Six alternative promoters are labeled. For transcripts, coding regions (CDSs) are in black and untranslated regions (UTRs) are in gray. Antp transcript annotation for J. coenia is from37.

Extended Data Fig. 7 Antp P2 mKO crispants in B. anynana.

Two guide RNAs co-injected efficiently induced long deletions around the conserved sequences (Fig. 4d middle panel) of Antp P2 in vivo, confirmed in pooled injected eggs (upper panel). The WT bands and mutant bands are denoted by black and red arrowheads, respectively. Genomic DNA was extracted from right hindwings of eight mKO crispants, and subjected to PCR and gel electrophoresis to detect potential long deletions (middle panel). Three crispants showing clear long deletions were genotyped via Nanopore amplicon sequencing. A dotted line separates left and right sides of the same individual. Purple arrowheads denote the wings used for genotyping, and red arrowheads denote guide RNA cut sites. Sequence coverage (Cov.) across the amplicon and major KO alleles are shown (lower panel).

Extended Data Fig. 8 Antp P1 mKO crispants in B. anynana.

Two guide RNAs co-injected efficiently induced long deletions around the conserved sequences (Fig. 4d right panel) of Antp P1 in vivo, confirmed in pooled injected eggs (top panel). The WT bands and mutant bands are denoted by black and red arrowheads, respectively. A dotted line separates left and right sides of the same individual. Purple arrowheads denote mutant phenotypes.

Extended Data Fig. 9 An Antp P1 mutant line in B. anynana.

A mutant line carrying a 252 bp deletion around the conserved region (Fig. 4d right panel) of Antp P1 was generated (Methods). The gel image shows clear differentiation of PCR bands across WT, mutant heterozygotes, and mutant homozygotes. The WT bands and mutant bands are denoted by black and red arrowheads, respectively. Representative wings for each genotype in both seasonal forms are shown. A dotted line separates dorsal (left) and ventral (right) sides of the same individual.

Extended Data Fig. 10 Antp P1 mutant heterozygotes do not exhibit significant changes in plasticity levels.

Changes in eyespot size plasticity levels were assessed (two-tailed) across sib-paired WT (P1+/P1+, n = 27 (WS) and 20 (DS) individuals) and mutant heterozygotes (P1+/P1Δ252, n = 30 (WS) and 33 (DS) individuals) in a two-way ANCOVA, indicated by a significant (padj<0.05) genotype (G) x temperature (T) interaction (padj: 0.72 (Rs), 0.78 (M1), 0.80 (M2), 0.88 (M3), 1.0 (Cu1), 0.90 (Cu2), 0.82 (Pc)). Lines with error bands represent mean values ± 95CI. Detailed sample sizes are summarized in Supplementary Table 10. Full statistical results are summarized in Supplementary Table 11. P values were adjusted for multiple comparisons using the Benjamini-Hochberg procedure. ns, not significant; *padj<0.05; **padj<0.01; ***padj<0.001.

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Tian, S., Lee, B., Banerjee, T.D. et al. A novel Hox gene promoter fuels the evolution of adaptive phenotypic plasticity in wing eyespots of satyrid butterflies. Nat Ecol Evol (2025). https://doi.org/10.1038/s41559-025-02891-5

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